黄清育，博士，研究员，博士生导师。福建省杰出青年基金获得者，中国科学院青年创新促进会会员，入选福建省和厦门市高层次人才及厦门市青年创新人才。2011年毕业于厦门大学生命科学学院，获理学博士学位。2011年10月至今在中国科学院城市环境研究所工作。2016年赴南丹麦大学访学一年。主要研究方向为环境污染物暴露的人群健康效应及毒理学机制。主持国家自然科学基金、福建省杰出青年基金、厦门市青年创新基金等科研课题10余项。已在Environmental Science & Technology、Environment International、Journal of Hazardous Materials等环境领域知名期刊上发表SCI论文80余篇，被引用1900余次；担任ES&T，EI，JHM等期刊审稿人。
环境污染与健康、环境毒理学、分子流行病学、组学、表观遗传学
1.Lu L, Hua W, Li F, Che Z, Tian M, Lu YY, Chi Q, Zhang J, Huang Q*. Arsenic exposure triggers nonalcoholic fatty liver disease through repressing S-adenosylmethionine-dependent histone methylation in rats. Environ. Sci. Technol. 2025, 59: 268-278.
2.Hua W, Han X, Li F, Lu L, Sun Y, Hassanian-Moghaddam H, Tian M, Lu Y, Huang Q*. Transgenerational effects of arsenic exposure on learning and memory in rats: Crosstalk between arsenic methylation, hippocampal metabolism, and histone modifications. Environ. Sci. Technol. 2024, 58: 6475-6486.
3.Han X, Alam MN, Cao M, Wang X, Cen M, Tian M, Lu Y, Huang Q*. Low levels of perfluorooctanoic acid exposure activates steroid hormone biosynthesis through repressing histone methylation in rats. Environ. Sci. Technol. 2022, 56: 5664-5672.
4.Tian M, Li H, Wu S, Xi H, Wang YX, Lu YY, Wei L, Huang Q*. Exposure to haloacetic acid disinfection by-products and male steroid hormones: An epidemiological and in vitro study. J. Hazard. Mater. 2024, 468: 133796.
5.Lu YY, Lu L, Ren HY, Hua W, Zheng N, Huang FY, Wang J, Tian M, Huang Q*. The size-dependence and reversibility of polystyrene nanoplastics-induced lipid accumulation in mice: Possible roles of lysosomes. Environ. Int. 2024, 185: 108532.
6.Lu YY, Cao M, Li F, Tian M, Ren H, Chi Q, Huang Q*. Atmospheric PM2.5 induce autophagy and autophagic flux blockage in HUVEC cells via ROS/TXNIP signaling: Important role of metal components. J. Hazard. Mater. 2023, 445: 130623.
7.Tian M, Wu S, Wang YX, Liu L, Zhang J, Shen H, Lu Y, Bao H, Huang Q*. Associations of environmental phthalate exposure with male steroid hormone synthesis and metabolism: an integrated epidemiology and toxicology study. J. Hazard. Mater. 2022, 436: 129213.
8.Huang Q*, Luo L, Han X, Li F, Zhang X, Tian M. Low-dose perfluorooctanoic acid stimulates steroid hormone synthesis in Leydig cells: Integrated proteomics and metabolomics evidence. J. Hazard. Mater. 2022, 424: 127656.
9.Han X, Tian M, Shliaha PV, Zhang J, Jiang S, Nan B, Alam MN, Jensen ON, Shen H, Huang Q*. Real-world particulate matters induce lung toxicity in rats fed with a high-fat diet: Evidence of histone modifications. J. Hazard. Mater. 2021, 416: 126182.
10.Tian M, Wang YX, Wang X, Wang H, Liu L, Zhang J, Nan B, Shen H, Huang Q*. Environmental doses of arsenic exposure are associated with increased reproductive-age male urinary hormone excretion and in vitro Leydig cell steroidogenesis. J. Hazard. Mater. 2021, 408: 124904.
11.Huang Q*, Liu L, Wu Y, Wang X, Luo L, Nan B, Zhang J, Tian M, Shen H. Seminal plasma metabolites mediate the associations of multiple environmental pollutants with semen quality in Chinese men. Environ. Int. 2019, 132: 105066.
12.Huang Q, Hu D, Wang X, Chen Y, Wu Y, Pan L, Li H, Zhang J, Deng F, Guo X, Shen H. The modification of indoor PM2.5 exposure to chronic obstructive pulmonary disease in Chinese elderly people: A meet-in-metabolite analysis. Environ. Int. 2018, 121: 1243-1252.
13.Liu S#, Huang Q#, Wu Y, Song Y, Dong W, Chu M, Yang D, Zhang X, Zhang J, Chen C, Zhao B, Shen H, Guo X, Deng F. Metabolic linkages between indoor negative air ions, particulate matter and cardiorespiratory function: A randomized, double-blind crossover study among children. Environ. Int. 2020, 138: 105663.
14.Liu S#, Huang Q#, Zhang X, Dong W, Zhang W, Wu S, Yang D, Nan B, Zhang J, Shen H, Guo X, Deng F. Cardiorespiratory effects of indoor ozone exposure associated with changes in metabolic profiles among children: A repeated-measure panel study. The Innovation 2021, 2: 100087.
15.Zhang Q, Tian M, Zhang X, Zhang X, Yang X, Lu YY, Li S, Liu L, Li J, Hassanian-Moghaddam H, Tang N, Huang Q*. Metabolic biomarkers linking urinary arsenic species to gestational diabetes mellitus: A cross-sectional study in Chinese pregnant women. Sci. Total Environ. 2023, 892: 164761.
16.Lu YY, Yang R, Cao M, Lu L, Zhu W, Hua W, Tian M, Sun Y, Huang Q*. Reversibility of polystyrene nanoplastics-induced disruption of testosterone biosynthesis in mice: The role of histone modifications. Environ. Pollut. 2025, 366: 125506.
17.Li F, Yang R, Lu L, Hua W, Sun Y, Tian M, Lu Y, Huang Q*. Comparative steroidogenic effects of hexafluoropropylene oxide trimer acid (HFPO-TA) and perfluorooctanoic acid (PFOA): Regulation of histone modifications. Environ. Pollut. 2024, 350: 124030.
18.Alam MN, Han X, Nan B, Liu L, Tian M, Shen H, Huang Q*. Chronic low-level perfluorooctane sulfonate (PFOS) exposure promotes testicular steroidogenesis through enhanced histone acetylation. Environ. Pollut. 2021, 284: 117518.
19.Huang Q, Xi G, Alamdar A, Zhang J, Shen H. Comparative proteomic analysis reveals heart toxicity induced by chronic arsenic exposure in rats. Environ. Pollut. 2017, 229: 210-218.
20.Alamdar A, Tian M, Huang Q*, Du X, Zhang J, Liu L, Shah STA, Shen H. Enhanced histone H3K9 tri-methylation suppresses steroidogenesis in rat testis chronically exposed to arsenic. Ecotoxicol. Environ. Saf. 2019, 170: 513-520.
1. 国家自然科学基金面上项目：代谢和组蛋白修饰共调控在砷暴露致学习记忆障碍中的作用机制研究（22076179），2021.1-2024.12.
2. 国家自然科学基金面上项目：表观遗传调控在PFCs干扰雄性生殖内分泌中的作用机制（21677142），2017.1-2020.12.
3. 国家自然科学基金青年基金项目：砷暴露对精子组蛋白修饰的影响及其男（雄）性生殖毒性机制研究（21307127），2014.1-2016.12.
4. 福建省杰出青年基金项目：基于“肠-代谢-睾丸”轴的全氟化合物雄性生殖毒性机制研究（2022J06033），2022.8-2025.8.
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6. 福建省自然科学基金面上项目：基于表观遗传（组蛋白修饰）的 PM2.5雄性生殖毒性机制研究（2015J01066），2015.4-2018.4.
7. 中国科学院青年创新促进会人才项目（2019305），2019.1-2022.12.
8. 中国科学院城市环境研究所青年人才领域前沿项目：慢性PM2.5暴露对机体内源性代谢物的影响及其诱发代谢紊乱相关疾病的风险研究（IUEQN201301），2013.9-2016.8.
9. 厦门市青年创新基金项目：代谢和表观遗传共调控在砷暴露致学习记忆障碍中的作用机制研究（3502Z20206091），2020.7-2023.6.
10. 宁波市自然科学基金项目：组蛋白修饰调控在重金属介导的雄性生殖毒性中的作用机制研究（2013A610189），2013.3-2015.3.
